Vitamin D Supplementation Linked to Telomere Length, Potentially Slowing Aging, Study Finds
LIMERICK, ireland – A recent study from the University of Augusta suggests a link between vitamin D supplementation and telomere length, raising the possibility that the vitamin could play a role in slowing the aging process. Telomeres, protective caps on the ends of chromosomes, shorten with age, and maintaining their length is associated with cellular health and longevity.
Researchers followed nearly 300 participants for a year, finding those taking 2,000 IU of vitamin D daily experienced a statistically notable increase in telomere length compared to a placebo group. The findings, published recently, build on previous research demonstrating a correlation between vitamin D deficiency and accelerated telomere shortening.
However, experts caution against interpreting the results as a green light for widespread high-dose supplementation. Some research indicates extremely long telomeres may actually increase disease risk, suggesting an optimal range remains unknown.
There is also no consensus on the ideal dosage. While the Augusta study utilized 2,000 IU daily, current recommended intakes are 600 IU for adults under 70 and 800 IU for those over 70.Other studies have suggested benefits from as little as 400 IU in preventing colds. Experts believe the optimal dose likely varies based on individual factors like existing vitamin D levels, nutrition, and nutrient interactions.”Even though these findings are exciting, its too early to start popping high-dose vitamin D in the hope of slowing aging,” says Dervla Kelly, Associate Professor of Pharmacology at the University of Limerick. “The strongest evidence for healthy aging still points to the basics: a balanced diet, regular exercise, quality sleep, not smoking and managing stress.”
Supplements remain a sensible choice for individuals deficient in vitamin D or at risk of poor bone health, supported by decades of established research. Scientists continue to investigate vitamin D’s complex role in cellular health and aging, viewing it as potentially one piece of a larger puzzle.
