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Title: Man Dies After Mistaking Cold Symptoms for Heart Attack – Testosterone Link

Ecuadorian TV Presenter⁤ Dies After Mistaking Heart Attack Symptoms for a Cold

Emilio⁢ Sueños,​ a popular 41-year-old television presenter ‍and model in Ecuador, recently died after experiencing rapidly worsening symptoms he initially believed were a common cold. He was admitted to the‍ intensive care unit of a Guayaquil hospital on October 1st,where​ doctors‍ diagnosed heart inflammation and hypertrophy ​linked to excessive testosterone use,according to‌ reports from⁤ The Sun and local media.

An acquaintance,⁣ Ivanna ⁢Melgar, shared ​that Sueños⁣ had complained of increasing cough and shortness of‌ breath in the⁢ days leading up to his⁢ hospitalization, even ‍sending a voice message stating, “It’s not the flu, but I can’t breathe.” reports indicate Sueños ⁤had been receiving hormone injections following previous cosmetic procedures. Complications⁣ arose due to remaining biopolymer material from a prior treatment on his buttocks, leading to injections being administered in⁤ his arm, which ultimately worsened⁢ his condition. Despite receiving blood transfusions‌ and⁣ detoxification treatment, he did not⁣ recover.

Sueños was a‍ well-known figure in Ecuadorian‌ broadcasting and a vocal advocate for⁤ the​ LGBTQ+ ⁤community. ‌Colleagues and fans have remembered⁤ him​ as a “luminous and sincere broadcaster.”

The dangers ⁣of Testosterone Abuse

His death highlights the potentially fatal cardiovascular risks associated‍ with high-dose testosterone ‍use.‌ while ‍testosterone supplementation is medically utilized to ⁢treat​ low hormone levels, self-governance of high doses or the use of anabolic‌ androgenic steroids (AAS) for cosmetic or muscle-building purposes can have severe consequences. AAS are synthetically produced versions of testosterone⁣ designed to‍ enhance muscle growth and strength.

Research ⁢consistently demonstrates‌ a strong link between AAS abuse and serious ‍heart⁢ problems. These ⁢include myocardial hypertrophy ⁢(enlargement of the heart⁢ muscle), myocardial fibrosis (scarring of the ⁣heart muscle), reduced⁢ heart function, arrhythmias (irregular heartbeats),‍ and ⁤an increased risk of heart failure.

A 2017 study published in ‍ Circulation, the journal of the American Heart Association,⁤ found that individuals using high-dose AAS exhibited increased left ventricular​ mass, myocardial⁣ fibrosis, and⁤ decreased contractile function, all ‍contributing to a higher risk of heart failure and arrhythmia.⁢

More recent research, published in the European Journal of Preventive Cardiology in⁢ 2024,⁣ showed that long-term AAS users experienced reduced contractility in both the left and right ventricles, along ​with ‍thickening of the⁤ heart walls.Some of these structural abnormalities persisted even after discontinuing AAS use.A 2023 review in Frontiers in Cardiovascular Medicine documented cases of⁤ dilated cardiomyopathy – a‌ weakening of ⁣the heart muscle – occurring in young adults without ⁤pre-existing heart conditions, ⁤with some instances resulting⁣ in irreversible damage due to myocardial fibrosis.

Conversely,medically supervised,low-dose testosterone⁢ supplementation ‌therapy does not appear to significantly increase the‌ risk of⁢ major cardiovascular events.⁣ Though, this finding is limited to treatment regimens ​administered ‌and monitored by qualified‍ medical professionals.

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